Meinig was well aware that bacteria was left behind after root canal therapy, leaking toxicity called endotoxins into the body, as he was an endodontist himself. However, he also stated that people do not have to become sick or die from focal infections, but to stop or prevent their occurrence, the bacteria and their toxins which arise from the dentin tubules or other sources must be eliminated.
George E Meinig Root Canal Cover Up.pdf
That was then and this is now. A lot has changed in the world of root canal therapy, especially in the last five years. New technologies have come onto the scene that will change the results possible and improve the health of not only teeth but the patient as well. Also, new protocols have been developed using these technologies that allow results not possible just 5 years ago. These technologies include:
What is revolutionary about the work of these specialists is that it addresses the latent bacteria left behind by traditional endodontics. All the developments above are recent, having taken place over the last 5 years or so, but research is starting to come out showing just how effective PIPS and SWEEPS are in eliminating even deep-seated bacteria in microtubules of root canal teeth. See my articles over the next few months highlighting some of these research papers.
Recommendation #1: In my experience, up to 40% of the root canals done in the past have some level of bacterial infection that is visible on imaging. So, if you have root canals and suspect that one or more of them may be infected, take a 3-D image to be sure. If you are not local to me, this can be set up at a lab in your area.
I have been sick for 60 years now a lot of contributions from inappropriate dentistry.. the most recent crisis was from a metal implant place in Jan 2016. In May 2016i developed breathlessness and extreme soreness/ pain under my left breast. Oxygen sats dropped to 90 and pain developed in nose and eye and ACTH and TSH levels failed Eventually a doctor in Germany advised removal of metal and roof canals Recovery is slow and only partial . Oxygen is 96 to 98 per cent most of time. But I hav sudden pain in site of implant and up to my eye and under my lung and down my leg. Lymph gathering under my eye and in ankle can you suggest what I can do next. I am Irish. I squeezed out some of fluid from under my eye about 2 weeks ago. It is v annoying It has also caused electric sensitivity and cost me around 20000 euro so far Many thanks for your work Mary doody
Hi Nicole, Sorry you are having so many problems with your teeth. I mentioned an endodontist in Los Angeles in my article that I recommend to patients. Did you check her out? Extraction may be an option for you, but that is not an easy road either, and partials are a compromise, especially when compared with your real teeth. I would definitely get a second opinion and see if your root canal teeth are truly infected.
Dr. George Meinig, a founding member of the American Association of Endodontists and dentist for the Twentieth Century Fox Studio, reveals the research which shows that a high percentage of chronic and degenerative diseases are made worse by root canal caused infections.
GUIGAND Martine (DCD), VULCAIN Jean-Marie (DCD, PhD), DAUTEL-MORAZIN Anne (DCD, BONNAURE-MALLET Martine (DCD, PhD), An ultrastructural study of root canal walls in contact with endodontic biomaterials, Étude, Journal of Endodontic, Vol. 23, No 5, 4 p., May 1997ALB 035.pdf
CAVALLERI Giacomo, URBANI Giacomo, DE FAZIO Pietro, PETRECCA Sergio, In vivo comparison between calcium hydroxide and calcium oxyde in the root canal medication, Article/Étude, J lt Endo, Vol. IV, Nr. 3 , 3 p., 1990ALB 036.pdf
OSORIO DIAZ Yosette (Odontologist), FAJARDO MARINO Fernando (Medical Dr.), Usage of traumeel on root canal treatment, Étude, Biological management of endodontics, 9 p.ALB 043.pdf
KEROSUO E., HAAPASALO M., LOUNATMAA K., RANTA H., RANTA K. (University of Kelsinki, Finland), Ultrastructure of a novel anaerobic gram-positive nonsporing rod from dental root canal, Étude, Scand J Dent Res, 1 p., Feb. 1988ALB 048.pdf
FUKUSHIMA H., YAMAMOTO K., HIROHATA K., SAGAWA H., LEUNG K.P., WALKER C.B. (University of Florida, Gainesville), Localisation and identification of root canal bacteria in clinically asymptomatic periapical pathosis, Étude, J Endod, 1 p., Nov. 1990ALB 054.pdf
BAUMGARTNER J.C., FALKLER W.A. Jr. (Walter Reed Army Medical Center, Washington, DC), Bacteria in the apical 5 mm of infected root canals, Étude, J Endod, 1 p., Aug. 1991ALB 056.pdf
MOLVEN O., OLSEN I., KEREKES K. (University of Bergen, Norway), Scanning electron microscopy of bacteria in the apical part of root canals in permanent teeth with periapical lesions, Étude, Endod Dent Traumatol, 1 p., Oct. 1991ALB 057.pdf
HORIBA N., MAEKAWA Y., ABE Y., ITO M., MATSUMOTO T., NAKAMURA H. (Aichi-Gakuin University, Nagoya, Japan), Correlations between endotoxin and clinical symptoms or radiolucent areas in infected root canals, Étude, Oral Surg oral med pathol, 1 p., Apr. 1991ALB 059.pdf
SUNDQVIST G. (University of Umea, Sweden), Associations between microbial species in dental root canal infections, Étude, Oral Microbiol Immunol, 1 p., Oct. 1992ALB 060.pdf
HASHIOKA K., YAMASAKI M., NAKANE A., HORIBA N., NAKAMURA H. (Aichi Gakuin University, Nagoya, Japan), The relationship between clinical symptoms and anaerobic bacteria from infected root canals, Étude, J Endod, 1 p., Nov. 1992ALB 061.pdf
GOMES B.P., LILLEY J.D., DRUCKER D.B. (University Dental Hospital of Manchester, UK), Clinical significance of dental root canal microflora, Étude, J Dent, 1 p., Jan. 1996ALB 062.pdf
HORIBA N., MAEKAWA Y., MATSUMOTO T., NAKAMURA H. (Aichi-Gakuin University, Nagoya, Japan), A study of the distribution of endotoxin in the dentinal wall of infected root canals, Étude, J Endod, 1 p., Jul. 1990ALB 063.pdf
SUNDQVIST G. (University of Umea, Sweden), Taxonomy, ecology, and pathogenicity of the root canal flora, Article, Oral Surg oral med pathol, 1 p., Oct. 1994,ALB 064.pdf
KIPIOTI A., NAKOU M., LEGAKIS N., MITSIS F., Microbiological findings of infected root canals and adjacent periodontal pockets in teeth with advancesperiodontitis, Étude, Oral Surg oral med pathol, 1 p., 1984ALB 069.pdf
UENO K., YOSHIHASHI M., SAWADA N., NAKAJIMA M., ARAKI K., SUDA H. (Tokyo Medical and Dental University), Cytotoxicity of anaerobic bacteria isolated from infected root canal, Étude, Kokubyo Gakkai Zasshi, 1 p., Sep. 1993ALB 073.pdf
ANDO N., HOSHINO E. (Niigata University, Japan), Predominant obligate anaerobes invading the deep layers of root canal dentin, Étude, Int Endod J, 1 p., Jan. 1990ALB 076.pdf
KOBAYASHI T., HAYASHI A., YOSHIKAWA R., OKUDA K., HARA K. (Niigata University, Japan), The microbial flora from root canals and periodontal pockets of non-vital teeth associated with advanced periodontitis, Étude, Int Endod J, 1 p., Mar. 1999ALB 078.pdf
Is mercury the only dental hazard that can create conditions favorable to autoimmune diseases? No. There are bacteria in root canals that favor destruction of the nervous system and many other systems, resulting in the creation of autoimmune reactions.
Price was concerned about the pathological bacteria found in nearly all root canal teeth of that time. He was able to transfer diseases harbored by humans from their extracted root canal teeth into rabbits by inserting a fragment of a root canal root under the skin in the belly area of a test rabbit. He found that root canal fragments from a person who had suffered a heart attack, when implanted into a rabbit, would cause a heart attack in the rabbit within a few weeks. Transference of heart disease could be accomplished 100 percent of the time. Some diseases transferred only 88 percent of the time, but the handwriting was on the wall.
Dr. Price discovered that root canals had within them bacteria capable of producing many diseases. They had no place in the body. Which is more important? The life of the tooth or the life of the patient? This is still the primary argument facing us today.
The unexplained increase in MS (8800 to 123,000) coincided with the advent of high copper amalgams. The increase in ALS in the same year is suggestive of the same cause. ALS also increased in 1991 as more root canals were performed. Statistical coincidence?
Our first DNA studies examined bacteria retrieved from crushed root tips. We can identify eighty-three different anaerobic bacterial species with DNA testing. Root canals contain fifty-three different species out of these eighty-three samples. Some are more dangerous than others, and some occur frequently, some occasionally. Selecting those that occur more than 5 percent of the time, we found:
Just out of curiosity, we tested blood samples adjacent to the removed teeth and analyzed them for the presence of anaerobic bacteria. Approximately 400 percent more bacteria were found in the blood surrounding the root canal tooth than were in the tooth itself. It seems that the tooth is the incubator. The periodontal ligament supplies more food, therefore higher concentration of bacteria.
But the winner in pathological growth was in the bone surrounding the dead tooth. Looking at bacterial needs, there is a smorgasbord of bacterial nutrients present in the bone. This explains the tremendous increase in bacterial concentration in the blood surrounding the root canal tooth. Try sterilizing that volume of bone. 2ff7e9595c
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